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ausblenden erkunden Chor crizotinib met Rand Pole Kabine

Activated MET acts as a salvage signal after treatment with alectinib, a  selective ALK inhibitor, in ALK-positive non-small cell lung cancer
Activated MET acts as a salvage signal after treatment with alectinib, a selective ALK inhibitor, in ALK-positive non-small cell lung cancer

Cyclosporine A sensitizes lung cancer cells to crizotinib through  inhibition of the Ca2+/calcineurin/Erk pathway - eBioMedicine
Cyclosporine A sensitizes lung cancer cells to crizotinib through inhibition of the Ca2+/calcineurin/Erk pathway - eBioMedicine

CRZ in MET-Rearranged NSCLC - Lung Cancer - 2016 American Society of  Clinical Oncology Annual Meeting - Oncology - Clinical Care Options
CRZ in MET-Rearranged NSCLC - Lung Cancer - 2016 American Society of Clinical Oncology Annual Meeting - Oncology - Clinical Care Options

Response to Crizotinib Re-administration After Progression on Lorlatinib in  a Patient With ALK-rearranged Non–small-cell Lung Cancer - Clinical Lung  Cancer
Response to Crizotinib Re-administration After Progression on Lorlatinib in a Patient With ALK-rearranged Non–small-cell Lung Cancer - Clinical Lung Cancer

Crizotinib in Patients With MET-Amplified NSCLC - Journal of Thoracic  Oncology
Crizotinib in Patients With MET-Amplified NSCLC - Journal of Thoracic Oncology

ADAM17-Dependent c-MET-STAT3 Signaling Mediates Resistance to MEK  Inhibitors in KRAS Mutant Colorectal Cancer: Cell Reports
ADAM17-Dependent c-MET-STAT3 Signaling Mediates Resistance to MEK Inhibitors in KRAS Mutant Colorectal Cancer: Cell Reports

Expanding treatment options in NSCLC patients with rare mutations: ALK,  ROS1, MET, BRAF - memoinOncology
Expanding treatment options in NSCLC patients with rare mutations: ALK, ROS1, MET, BRAF - memoinOncology

Crizotinib is an Orally Active, ATP-Competitive ALK and c-Met inhibitor -  Network of Cancer Research
Crizotinib is an Orally Active, ATP-Competitive ALK and c-Met inhibitor - Network of Cancer Research

MET mutations identified following crizotinib treatment. (A–C) The... |  Download Scientific Diagram
MET mutations identified following crizotinib treatment. (A–C) The... | Download Scientific Diagram

Crizotinib exhibits antitumor activity by targeting ALK signaling not c-MET  in pancreatic cancer. - Abstract - Europe PMC
Crizotinib exhibits antitumor activity by targeting ALK signaling not c-MET in pancreatic cancer. - Abstract - Europe PMC

Safety and Tolerability of c-MET Inhibitors in Cancer | SpringerLink
Safety and Tolerability of c-MET Inhibitors in Cancer | SpringerLink

Downregulation of MET in H1993 cells, effects of crizotinib on EGFR... |  Download Scientific Diagram
Downregulation of MET in H1993 cells, effects of crizotinib on EGFR... | Download Scientific Diagram

Crizotinib, a MET inhibitor, prevents peritoneal dissemination in  pancreatic cancer
Crizotinib, a MET inhibitor, prevents peritoneal dissemination in pancreatic cancer

Antitumor activity of crizotinib in lung cancers harboring a MET exon 14  alteration | Nature Medicine
Antitumor activity of crizotinib in lung cancers harboring a MET exon 14 alteration | Nature Medicine

Crizotinib hydrochloride | c-Met 阻害剤
Crizotinib hydrochloride | c-Met 阻害剤

cに応じられた阻害 | c-Met Inhibition
cに応じられた阻害 | c-Met Inhibition

Crizotinib-resistant MET mutations in gastric cancer patients are sensitive  to type II tyrosine kinase inhibitors | Future Oncology
Crizotinib-resistant MET mutations in gastric cancer patients are sensitive to type II tyrosine kinase inhibitors | Future Oncology

JCM | Free Full-Text | The Role of MET Inhibitor Therapies in the Treatment  of Advanced Non-Small Cell Lung Cancer | HTML
JCM | Free Full-Text | The Role of MET Inhibitor Therapies in the Treatment of Advanced Non-Small Cell Lung Cancer | HTML

Crizotinib inhibits activation of MET pathway caused by MET extracellular  SEMA domain duplication - Lung Cancer
Crizotinib inhibits activation of MET pathway caused by MET extracellular SEMA domain duplication - Lung Cancer

Response to Crizotinib in a Patient With Lung Adenocarcinoma Harboring a MET  Splice Site Mutation - Clinical Lung Cancer
Response to Crizotinib in a Patient With Lung Adenocarcinoma Harboring a MET Splice Site Mutation - Clinical Lung Cancer

Crizotinib (PF-02341066) | ALK/c-Met/ROS1阻害剤 | MedChemExpress
Crizotinib (PF-02341066) | ALK/c-Met/ROS1阻害剤 | MedChemExpress

Antitumor activity of crizotinib in lung cancers harboring a MET exon 14  alteration | Nature Medicine
Antitumor activity of crizotinib in lung cancers harboring a MET exon 14 alteration | Nature Medicine

Effect of Crizotinib on c-MET expression in c-MET altered cancer cells....  | Download Scientific Diagram
Effect of Crizotinib on c-MET expression in c-MET altered cancer cells.... | Download Scientific Diagram

Activated MET acts as a salvage signal after treatment with alectinib, a  selective ALK inhibitor, in ALK-positive non-small cell lung cancer
Activated MET acts as a salvage signal after treatment with alectinib, a selective ALK inhibitor, in ALK-positive non-small cell lung cancer

PLOS ONE: Ability of the Met Kinase Inhibitor Crizotinib and New Generation  EGFR Inhibitors to Overcome Resistance to EGFR Inhibitors
PLOS ONE: Ability of the Met Kinase Inhibitor Crizotinib and New Generation EGFR Inhibitors to Overcome Resistance to EGFR Inhibitors

Characterization of MET exon 14 alteration and association with clinical  outcomes of crizotinib in Chinese lung cancers - Lung Cancer
Characterization of MET exon 14 alteration and association with clinical outcomes of crizotinib in Chinese lung cancers - Lung Cancer